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. 2016 Dec 27;114(2):E245–E254. doi: 10.1073/pnas.1613199114

Fig. 4.

Fig. 4.

BRC1, HB21, HB40, and HB53 promote ABA accumulation via NCED3. (A and B) NCED3 mRNA levels analyzed by quantitative PCR measured in axillary buds of wild-type plants and hb21 hb40 hb53-1 mutants (A) and brc1 mutants (B) treated with W or W+FR light for 8 h. (C) NCED3 mRNA levels in 7-d-old GUSind, HA:BRC1ind, HA:HB21ind, HA:HB40ind, and HA:HB53ind seedlings after an 8-h treatment with 10 µM estradiol. (D) ABA levels measured in estradiol-treated seedlings of the genotypes in C. (E) Branching phenotype of wild-type and hb21 hb40 hb53-2 plants treated with W or W+FR light and 50 µM ABA or mock (control) for 2 wk after bolting (n = 28). Error bars show the SEM of three biological replicates. Asterisks show significant differences (P < 0.05; student’s t-test) between control and treated plants. Letters denote significant differences (P < 0.05; one-way ANOVA) among means.