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. 2016 Sep 14;311(6):R1135–R1148. doi: 10.1152/ajpregu.00261.2016

Fig. 1.

Fig. 1.

Bco1−/− loss reduces seminal vesicular and prostatic weight as a percentage of body weight. A: anterior (AP), dorsolateral (DLP), and ventral prostate (VP) lobe weight percentages of body weight are shown. B: total prostate weight as a percentage of body weight is shown. Seminal vesicles (SV) (C) and testes (D) are also shown as a percentage of total body weight (%BW). E: total body weight. Data are presented as group mean %BW ± SE; n = 15, *P < 0.05, ***P < 0.001. F: PCR genotyping confirms Bco1 disruption in several tissues of male Bco1−/− mice. Lane 1, DNA ladder; lane 2, tail snip from Bco1−/− breeding dam; lane 3, tail snip from wild-type (WT) breeding dam (Jackson Laboratories); lane 4, liver sample from male Bco1−/− study animal; lane 5, testicle sample from Bco1−/− study animal; lane 6, AP sample from Bco1−/− study animal. WT and Bco1−/− band sizes correspond to those reported by Hessel et al. (33). WT: 351 base pairs (bp); Bco1−/−: 588 bp.