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. Author manuscript; available in PMC: 2017 Jan 23.

Published in final edited form as: Biochem J. 2008 Oct 15;415(2):309–316. doi: 10.1042/BJ20080973

Schematic representation of proline metabolism in ASE mitochondria

Panel A: Proline metabolism as described by [42–44, 47, 71].

Panel B: Proline metabolism expanded and modified according to the experimental results found in this study.

Inhibitors are shown in italics. 1, Δ¹-pyrroline-5-carboxylate reductase; 2, Δ¹-pyrroline-5-carboxylate dehydrogenase; 3, Glutamate-pyruvate transaminase; 4, ketoglutarate dehydrogenase; 5, succinylCoA synthetase; 6, succinate dehydrogenase; 7, fumarate reductase; 8, malic enzyme; 9, malate dehydrogenase; 10, citrate synthase; 11, aconitase; 12, isocitrate dehydrogenase; 13, pyruvate dehydrogenase; 14, aspartate-oxaloacetate transaminase; 15, glutamate dehydrogenase.

Schematic representation of proline metabolism in ASE mitochondria

Panel A: Proline metabolism as described by [42–44, 47, 71].

Panel B: Proline metabolism expanded and modified according to the experimental results found in this study.

Inhibitors are shown in italics. 1, Δ¹-pyrroline-5-carboxylate reductase; 2, Δ¹-pyrroline-5-carboxylate dehydrogenase; 3, Glutamate-pyruvate transaminase; 4, ketoglutarate dehydrogenase; 5, succinylCoA synthetase; 6, succinate dehydrogenase; 7, fumarate reductase; 8, malic enzyme; 9, malate dehydrogenase; 10, citrate synthase; 11, aconitase; 12, isocitrate dehydrogenase; 13, pyruvate dehydrogenase; 14, aspartate-oxaloacetate transaminase; 15, glutamate dehydrogenase.