Fig 9. Glucose-ABL1-TOR signaling mediates cell cycle progression and quiescence during appressorium formation.

Models of cell cycle tuning by the glucose-ABL1-TOR signaling axis during WT appressorial cell differentiation under (A) nutrient-free conditions and (B) in the presence of glucose or the absence of ABL1 function. Cell cycle phase lengths were determined from data in Figs 2, 4 and 6 and S8 Fig. Autophagy at G2 in (A) might yield glutamine (Gln), glucose (Glc) or ATP to activate TOR.