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. Author manuscript; available in PMC: 2017 Jan 25.
Published in final edited form as: Alcohol Clin Exp Res. 2010 Mar 10;34(5):886–896. doi: 10.1111/j.1530-0277.2010.01161.x

Figure 1.

Figure 1

Effects of chronic ethanol intake on the expression of OX in the PFLH, as measured by qRT-PCR (Experiment 1). In the 2% ethanol drinking group (a) three groups of rats (n=5/group), water, 0.25 g/kg/day and 0.75 g/kg/day ethanol drinking animals were examined. The data (mean ± SEM) revealed a significant reduction in expression of OX in the 0.75 g/kg/day ethanol group [F(2,15) =30.8, p<0.001]. In the 9% ethanol drinking group (b) three groups of rats (n=5/group), water, 1.0 g/kg/day and 0.75 g/kg/day ethanol drinking animals were examined. The data (mean ± SEM) revealed a significant reduction in expression of OX in the 1.0 g/kg/day and 2.5 g/kg/day ethanol groups [F(2,15) = 41.5, p<0.001]. This effect was statistically significant in the 0.75 g/kg, 1.0 g/kg and 2.5 g/kg ethanol drinking groups compared to their respective water group (*p<0.001), and the suppression in the 2.5 g/kg/day ethanol drinkers was significantly greater than that in the 1.0 g/kg ethanol drinkers (** p<0.001).