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. 2017 Jan 26;10:4. doi: 10.1186/s13072-017-0111-y

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© The Author(s) 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Differentially methylated regions associated with fetal smoking exposure. a Manhattan plot shows the most significant smoking exposure DMRs identified between smoking-exposed and unexposed fetal cortical samples, red line; adjusted p value = 0.05. DNA hypomethylation of fetal cortical samples exposed to maternal smoking was found within the promoter regions of the two most significant smoking exposure DMRs b SDHAP3 and c GNA15. CGI CpG Island. Gene expression of d SDHAP3 and e GNA15 shows temporal up-regulation in the fetal cortex, particularly in smoking exposed