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. 2017 Jan 26;36:20. doi: 10.1186/s13046-016-0485-x

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — NNRTIs are effective to restrain HERV-K activation and induce apoptosis in TVM-A12 CD133+ cells. Effects of NNRTIs on the expansion and maintenance of CD133+ cells in TVM-A12 (a) and TVM-A12-CD133+ (b) cell lines analyzed by flow cytometry analysis. Effects of NNRTIs on apoptosis levels in TVM-A12 (c) and TVM-A12-CD133+ (d) analyzed by flow cytometry analysis after nuclei staining with propidium iodide. Relative mRNA expression of HERV-K env gene (e) and CD133 (f) analyzed by Real-time PCR. Data represent the results of three independent experiments