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. Author manuscript; available in PMC: 2017 Feb 1.
Published in final edited form as: Circ Cardiovasc Qual Outcomes. 2016 May 24;9(4):432–440. doi: 10.1161/CIRCOUTCOMES.116.002614

Table 1.

Characteristics of Follow-Up Cohort

n %
EGA, wk, median (IQR) 39 (37, 39)
  <34 wk EGA 4 3.0
  34 0/7–36 6/7 wk EGA 18 13.6
  37 0/7–39 6/7 wk EGA 90 68.2
  ≥40 0/7 20 15.2
Birth weight, g, median (IQR) 3110 (2730, 3510)
Discharge age, mo, median (IQR) 1 (1, 2)
Sex (% female) 57 43.1
Race
  White 115 87.1
  Black 13 9.8
  Hispanic 12 9.0
  Other 4 3.0
Genetic syndrome 31 23.4
Trisomy 21 14 10.6
Failed ABR 8 7.0
Seizures on EEG 2 1.5
Discharge on full oral feeds 62 46.9
Neuroimaging
  Not performed 23 17.4
  Read as normal 56 42.4
  Read as severe neural injury* 17 12.9
  Other types of neuroimaging findings 36 27.3
Cardiac diagnosis
  Hypoplastic left heart 25 18.9
  Tetralogy of Fallot 14 10.6
  Truncus arteriosus 3 2.3
  Tricuspid atresia 6 4.5
  Transposition of the great vessels 11 8.3
  Total anomalous pulmonary venous return 5 3.8
  Pulmonary artery, critical pulmonary stenosis 5 3.8
  Aortic stenosis, atresia, coarctation 34 25.8
  Atrioventicular septal defect 16 12.1
  Other 13 9.8

ABR indicates auditory brainstem response; EEG, electroencephalogram; EGA, estimated gestational age at birth; IQR, interquartile range (25th, 75th).

*

Severe neural injury defined as intraventricular hemorrhage grades III or intraventricular hemorrhage, periventricular leukomalacia, diffuse ischemia; all severe findings documented on magnetic resonance imaging or computed tomography.

Intraventricular hemorrhage grades I or II, partial agenesis of the corpus callosum, mineralizing vasculopathy of noninfectious origin, small subdural hematomas.