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. Author manuscript; available in PMC: 2018 Feb 1.
Published in final edited form as: Mol Cancer Res. 2016 Nov 17;15(2):165–178. doi: 10.1158/1541-7786.MCR-16-0085-T

Figure 5.

Figure 5

Potential LSR transcript variants. A, LSR transcripts α-ι were identified from online databases and are shown with putative structural and protein-binding domains. Most LSR transcript variants appear to be the result of alternative splicing of particular exons. Ig-like – immunoglobulin-like; NLS – nuclear localization sequence; TM – transmembrane; C – cysteine-rich; SDR – short-chain dehydrogenase/reductase. B, Fourteen cell lines were examined for the expression of LSR (top panel) and a GAPDH loading control (bottom panel) by western blot analysis. Different cell lines expressed LSR at varying levels and at different molecular weights when using a polyclonal antibody that recognizes all predicted isoforms of LSR. Immunoblots are representative of a minimum of three independent experiments.