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. 2017 Feb 6;17:105. doi: 10.1186/s12885-017-3101-3

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Evaluation of cell proliferation by CCK-8 assays showed that down-regulation of CLASP2 dramatically inhibited the cell proliferation rate of CRL 1749 cells (a), whereas overexpression of CLASP2 significantly promoted the growth of HTB9 cells (b). Clonogenic formation assay found that CLASP2 increased bladder cancer cells proliferation (c) (**P <.01, *P <0.05)