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. 2017 Feb 8;10:32. doi: 10.3389/fnmol.2017.00032

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Copyright © 2017 Wang, Xiong, Wan, Duan, Bai and Zu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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BMP9 acted via a canonical Smad-dependent signaling pathway in APP/PS1 mice. (A) Representative western blots of pSmad1/5/8, Smad1/5/8, Smad4, pERK1/2, ERK1/2, pp38 mitogen-activated protein kinase (MAPK), p38 MAPK and GAPDH in the hippocampus homogenates from APP/PS1 mice treated with vehicle or BMP9. (B) Densitometric analyses of the immunoreactivities to the antibodies shown in the previous panel. Values are represented as the mean ± SD. Student’s t test. *p < 0.05, n = 6.