sod-1 |
|
|
sod-2 |
|
Accelerated wound healing [59]
Increased or no effect on
lifespan [66], [67], [94], [95]
Decreased or no effect in resistance to oxidative stress-inducing agents [66], [67], [94], [95]
|
sod-3 |
|
Accelerated wound healing [59]
Increased or no effect on lifespan [66], [67], [94], [95]
Decreased, increased or no effect in resistance to oxidative stress-inducing agents [66], [67], [94], [95]
|
sod-4 |
Superoxide anion to hydrogen peroxide
Nervous system, intestine, somatic gonad/Extracellular and membrane bound [16], [97], [98]
|
|
sod-5 |
|
|
prdx-2 |
Hydrogen peroxide reduction to water
Neurons, intestine and reproductive system/Cytoplasmic [32], [98], [99]
|
Decreased lifespan [32], [100]
Increased resistance to arsenite and cadmium but increased sensitivity to hydrogen peroxide [32]
Defective feeding response to light [12]
Reduced insulin secretion [87]
Defect in H2O2-induced potentiation of avoidance behaviour [13]
Reduced fecundity and small size [32,99]
Impaired PMK-1 activation by arsenite and metformin [32,77]
|
prdx-3 |
|
Decreased or no effect on lifespan (RNAi) [69], [101]aParaquat sensitive (RNAi) [101]a
Reduced fitness (RNAi) [69]a
|
prdx-6 |
|
|
ctl-1 |
|
|
ctl-2 |
|
|
ctl-3 |
|
|
bli-3 |
Hydrogen peroxide production and reduction
Hypodermis, intestine and pharynx/Subcellular localization not determined [18], [104]
|
Increased pathogen susceptibility [104]
Defective collagen cross-linking (RNAi) [18], [20]a
Blistered and molting defects [20]
|
mlt-7 |
|
|
skpo-1 |
|
|
glb-12 |
Superoxide anion production
Somatic reproductive system, vulva, head and tail neurons/Plasma membrane [16]
|
|