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. 2016 Dec 30;427(1):177–185. doi: 10.1007/s11010-016-2909-5

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 3 — MiR-192 and -218 mediated expression of ALCAM and N-cadherin on SFMSCs treated with gliomas-CM. a mRNA expression of ALCAM in SFMSCs was greater than that in SFM following treatment with glioma-CM. b N-cadherin expression was significantly upregulated in SFMSCs with CM from two gliomas. c Expression of miR-192 and -218 was downregulated in SFMSCs by gliomas-CM treatment. d The sequences of miR-192 and -218 showed that they can bind to regulate expression of target genes. Luciferase assay revealed that each target gene was regulated by miR-192 and -218, respectively. Data are expressed as the mean ± SD, *P < 0.05