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. 2016 Aug 23;7(38):62559–62571. doi: 10.18632/oncotarget.11537

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Copyright: © 2016 Jung et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Cells were stimulated with H₂O₂ in the absence or presence of NAC pretreatment for 1 h. H₂O₂ treatments were done for indicated times and analysis of intracellular ROS levels was performed for parental HCT116 p53 +/+ A., HCT116 p53 +/+ HA & HA-SOCS1 B., and sh & sh-SOCS1 C.