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. 2017 Feb 12;12:70–81. doi: 10.1016/j.redox.2017.02.006

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© 2017 Published by Elsevier B.V.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 2 — Changes in HIF-1α and its downstream targets NOX-4, HO-1 and iNOS in human atherosclerotic plaques. Representative western blots and relative densitometric analysis of Α. HIF-1α/β-tubulin B. NOX-4/β-actin C. Immunohistochemical (IHC) analysis of NOX-4 in serial sections from stable (n=8) and unstable plaques (n=8) D. HO-1/β-actin E. iNOS/β-actin F. Immunohistochemical (IHC) analysis of iNOS in serial sections from stable (n=8) and unstable plaques (n=8) (*p<0.05 vs Unstable). For IHC purposes, frames in 200x magnification pictures represent the areas of 400x magnification.