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. 2016 Dec 23;292(6):2531–2541. doi: 10.1074/jbc.M116.760256

FIGURE 4.

FIGURE 4.

POPS in acceptor vesicles does not stimulate C1P transfer and POPS itself is not transferred. Traces for a and b represent AV-C1P emission intensity measured at 415 nm as a function of time resulting from the loss of AV-SL/Per-PC FRET as AV-SL is transferred to POPC vesicles. The presence of POPS (10 mol %) in POPC receiver (acceptor) vesicles (10-fold excess) does not stimulate AV-C1P transfer by ACD11 (a) or CPTP (b). c, traces represent AV-PS emission intensity measured at 415 nm as a function of time resulting from the loss of AV-PS/Per-PC FRET. The AV-PS transfer to POPC receiver (acceptor) vesicles (10-fold excess) by ACD11, CPTP, and GLTP is virtually nil.