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. 2017 Feb 21;7:42926. doi: 10.1038/srep42926

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Copyright © 2017, The Author(s)

This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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(a) Schematic diagram indicating the position of RTN on a coronal mouse brain slice. Modified from Allen Brain Atlas. (http://atlas.brain-map.org/#atlas=1&plate=100960236&structure=549&x=5280&y=3744.0000697544647&zoom=-3&resolution=11.97&z=6). Relative expression of AMPAR subunit GluA4 (b), and NMDAR subunits NR1 (c), NR2A (d), NR2B (e) in stargazer RTN; left panel: representative cropped WB images of protein of interest, as well as VGlut1, and β-actin in NE (non-epileptic) and E (epileptic) RTN, right panel: scatter-column combination chart depicting relative epileptic expression of GluA4 (n = 8 NE, n = 7 E; ****p ≤ 0.0001), NR1 (n = 12 NE, n = 8 E; p > 0.05), NR2A (n = 12 NE, n = 10 E; p > 0.05) and NR2B (n = 14 NE, n = 11 E; p > 0.05).