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. 2017 Jan 31;114(7):1666–1671. doi: 10.1073/pnas.1621485114

Table 1.

Compiled quantitation data from in vitro, cell culture, and in vivo experiments on nsP3 WT and mutants of CHIKV 181/25 strain

CHIKV strain ADP-ribose KD, μM Hydrolase activity, % Virus progeny Sequencing Replication (NSC-34), % Mortality in mice, % Mean days of death
WT 22.9 ± 3.7 100 Yes 100 100 2.5
Category 1
 D10A NDB 16.1 ± 2.3 No Reverted to WT N/A N/A N/A
 G32A 21.0 ± 3.7 75.3 ± 9.3 Yes 7 58 6.9
 G32S 42.9 ± 17.7 48.0 ± 16.0 Yes 4 4 Undefined
 G32E NDB 3.8 ± 9.1 No Reverted to WT N/A N/A N/A
 T111A 71.4 ± 11.5 64.1 ± 4.0 Yes <1 44 5.3
 G112E NDB 3.7 ± 2.5 No Reverted to WT N/A N/A N/A
 R144A 64.9 ± 14.9 96.2 ± 2.3 No Reverted to WT N/A N/A N/A
Category 2
 Y114A 4.84 ± 0.69 40.6 ± 4.2 Yes 16 (Delayed) 75 5.3
 G32E V113R Y114N 6.46 ± 1.25 15.0 ± 7.2 No Reverted to either G32A V113R Y114N or V113R Y114N N/A N/A N/A

Hydrolase activity is calculated as a percentage of nsP3 WT activity after subtraction of buffer control. Replication rate is defined as the number of plaques in a mutant divided by the number of plaques in WT. CHIKV 181/25E2 I12T R82G and mutant derivatives were used in mouse experiments. N/A, not available; NDB, no detectable binding.