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. 2017 Feb 28;11:43. doi: 10.3389/fncel.2017.00043

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Copyright © 2017 Zhang, Wu, Shi, Xu, Chu, Cui and Qiu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The ethanol-induced inhibition of CS activity is persistent in the presence of an N-methyl-D-aspartate receptors (NMDAR) blocker. (A) Representative traces showing the properties of the CS-evoked inward currents in the presence of ACSF, D-(-)-2-Amino-5-phosphonopentanoic Acid (D-APV; 250 μM) or a mixture of D-APV + ethanol (300 mM), and recovery. (B,C) Bar graphs showing the effect of ethanol on the spikelet number of CSs (B) and the AUC of CSs in the presence of ACSF, D-APV (250 μM) or a mixture of D-APV (50 μM) + ethanol, and recovery. n = 6 cells per group. *P < 0.05, vs. ACSF.