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. 2017 Feb 28;11:43. doi: 10.3389/fncel.2017.00043

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Copyright © 2017 Zhang, Wu, Shi, Xu, Chu, Cui and Qiu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Ethanol fails to inhibit the CS-evoked change in the membrane potential of PCs in the presence of a CB1 receptor agonist, WIN55212-2. (A) Representative traces showing the spontaneous CS-evoked membrane potential in treatments of ACSF, WIN55212-2 (5 μM) and ethanol + WIN55212-2. (B,C) Bar graphs showing the normalized spikelet number (B) and AUC (C) of the CS-evoked potential in treatments of ACSF, WIN55212-2 and ethanol + WIN55212-2. (D) Pooled data showing the normalized pause in SSs in treatments of ACSF, WIN55212-2 and ethanol + WIN55212-2. (E) Bar graph showing the normalized amplitude of the AHP in treatments of ACSF WIN.55212-2 and ethanol + WIN55212-2. n = 6 cells per group. *P < 0.05, vs. ACSF.