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Suppression of HDAC and PI3K signaling by CUDC-907 in CRC cells. Immunoblotting of H3K9ac in CUDC-907-treated HCT116 cells (a), and Akt (Ser473) in CUDC-907-treated HCT116 cells post insulin activation of the PI3K pathway (b). β-Actin was used as loading control
