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. 2017 Feb 28;17:161. doi: 10.1186/s12885-017-3139-2

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — MT1H plays a pivotal role in HCC cell proliferation. a Expression of MT1H-Flag in indicated cells. Protein extracts from indicated cells were immunoblotted with antibody for Flag. α-Tubulin as a loading control. b HepG2-Vector, HepG2-MT1H, Hep3B-Vector, and HepG3B-MT1H cells was measured by MTT response. Error bars indicate standard deviation (Student’s t-test; *, P < 0.05). c Colony number of MT1H overexpressing HepG2 and Hep3B cells was significantly decreased as compared with respective vector-control. Error bars indicate standard deviation (Student’s t-test; *, P < 0.05). d Edu incorporation analysis indicates that MT1H suppresses the proliferation of HCC cells. Error bars indicate standard deviation (Student’s t-test; *, P < 0.05)