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. Author manuscript; available in PMC: 2017 Feb 28.
Published in final edited form as: Prog Mol Biol Transl Sci. 2015 Nov 17;137:1–40. doi: 10.1016/bs.pmbts.2015.10.017

Table 1.

Effects of MEK inhibitors on drugs-induced behaviors

Drugs MEK inhibitors (dose, area) Behavioral effects References
None SL327 (50 mg/kg, i.p.) ↑ basal locomotor activity 152
SL327 (50–100 mg/kg, i.p.) ↓ basal locomotor activity 92,156,254,255
PD98059 (50 μM, continuous infusion into the PFC) ↑ basal locomotor activity↓ 154
Cocaine SL327 (50 mg/kg, i.p.) ↓ acute cocaine-induced locomotion 78
SL327 (30 mg/kg, i.p.); PD98059 (10 μM, VTA) ↓ development of locomotor sensitization (inhibitors were injected/infused before each cocaine injection) 92,94
SL327 (40 mg/kg, i.p.); PD98059 (2 μg) or U0126 (1 μg, NAc) ↓ expression of locomotor sensitization (inhibitors were injected/infused before cocaine challenge) 80,93
SL327 (30 mg/kg, i.p.) ↓ conditioned locomotor response (inhibitor was injected before each cocaine injection during conditioning) 92
SL327 (50 mg/kg, i.p.); U0126 (0.1 μg, VTA) ↓ development of CPP (inhibitors injected/infused before each cocaine injection during conditioning) 78,100
U0126 (1 μg, NAc core) ↓ expression of CPP (inhibitor was infused before CPP test) 107
SL327 (30 mg/kg, i.p.); PD98059 (2 μg ) or U0126 (1 μg, NAc core); U0126 (1 μg, BLA) ↓ context- and cocaine priming-induced expression of CPP and
↓ context-induced reinstatement after SA by impairing memory reconsolidation (inhibitors were injected/infused either before or after reconsolidation phase)
82,107,112,113
U0126 (1 μg, CeA) ↓ context+cues-induced relapse after abstinence from SA (inhibitor was infused before relapse testing) 116
U0126 (1 μg, VTA) ↓ BDNF/GDNF-enhanced relapse by context+cues after abstinence from SA (infusions were conducted immediately after the end of the last SA session) 118,119
U0126 (0.5 μg, dmPFC) ↓ BDNF’s inhibitory effect on context-, cues- and cocaine priming-induced drug seeking after abstinence/extinction of SA (infusions were conducted immediately after the end of the last SA session) 121
Amphetamine SL327 (50–100 mg/kg, i.p.) ↓ acute amphetamine-induced locomotion 78,140,141
PD98059 (50 μM, continuous infusion into the PFC) ↑ acute amphetamine-induced locomotor activity 154
SL327 (40 mg/kg, i.p.) ↓ expression of locomotor sensitization (inhibitors were injected/infused before amphetamine challenge) 80
SL327 (30 mg/kg, i.p.) ↓ conditioned locomotor response (inhibitor was injected before each amphetamine injection during conditioning) 92
PD98059 (2.5 μg, NAc ) ↓ development of intra-NAc amphetamine-induced CPP (inhibitor was infused before or after each intra-NAc amphetamine infusion during conditioning) 160
PD98059 (2 μg, NAc ) ↓ expression of amphetamine-CPP (inhibitor was infused before CPP testing) 180
Marijuana (THC) SL327 (50 mg/kg, i.p.) ↓ development of THC-induced locomotion tolerance ( inhibitor was injected before each THC administration) 200
SL327 (50 mg/kg, i.p.) ↓ development of THC-CPP (inhibitor was injected before each conditioning session) 196
Alcohol PD98059 (30 or 90 μg, i.c.v ) ↓ development of ACD-CPP (inhibitor was infused before each conditioning session) 253
SL327 (30 mg/kg, i.p.) ↑ ethanol SA (inhibitor was injected before SA session) 255
U0216 (0.5 μg, VTA) ↓ GDNF’s inhibitory effect on ethanol SA (infusions were conducted before SA session) 259

i.p. = intraperitoneal injection; i.c.v = intracerebroventricular infusion; ↑ = enhancing effect; ↓ = inhibiting effect