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. 2017 Feb 28;23(8):1375–1386. doi: 10.3748/wjg.v23.i8.1375

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©The Author(s) 2017. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Lack of heme oxygenase-1 reverses the protective role of hydrogen-rich water in dextran sulfate sodium-induced inﬂammatory bowel disease. Mice were sacrificed, and the colons were rapidly removed and processed for analysis on the 7^th d after inﬂammatory bowel disease (IBD) modeling. A: The representative pictures of the colon in each group; B: The macroscopic score of the colon in each group; C: The length of the colon in each group; D: The thickness of the colon in each group; E: Hematoxylin-eosin staining of colon tissues (magnification ×100). n = 6, mean ± SEM, ^aP < 0.05 vs dextran sulfate sodium (DSS) group; ^bP < 0.05 vs DSS + HRW group. HO-1: Heme oxygenase-1; HRW: Hydrogen-rich water; HE: Hematoxylin and eosin.