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. 2017 Mar 3;7:43773. doi: 10.1038/srep43773

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Copyright © 2017, The Author(s)

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(a) A western blot depicting VDR expression in SNU308-VDRsi (VDR knockdown) and SNU308-COLsi (mock knockdown) cells. Actin was used as the loading control. (cropped). (b) After 7 days of treatment, MART-10 repressed SNU308-VDRsi cell growth, which was less than that noted in SNU308 cells.(*p < 0.05, **p < 0.01). (c)The NGAL protein expression in SNU308-VDRsi cells was not influenced by 10⁻⁸ M MART-10. (d) The NGAL reporter activity was inhibited by MART-10 in a dose dependent manner. (*p < 0.05). (e) VDR knockdown in SNU308 cells partly abolished MART-10-induced inhibition of NGAL reporter activity. (*p < 0.05 **p < 0.01).