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. 2017 Mar 6;11:57. doi: 10.3389/fncel.2017.00057

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Copyright © 2017 Wang, He, Guo, Cheng, Wang, Wang, Feng, Vreugdenhil and Lu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The effect of protein kinase A (PKA) inhibitor on the nicotinic modulation of γ oscillations. (A,C) The extracellular field potential recordings from the CA3 stratum pyramidale, for the kainate alone (control, trace taken at a time point that γ oscillation was stable for 60 min after kainate), in the presence of PKA inhibitor, 10 μM H89 (40 min after H89), and after additional application of 1 μM nicotine (30 min after nicotine; A) or 100 μM nicotine (30 min after nicotine; C). (B,D) The power spectra corresponding to (A,C). (E) γ power as % of baseline γ power for control, in the presence of H89, and with additional application of 1 μM nicotine (n = 12) or 100 μM nicotine (n = 8).