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. 2017 Jan 12;173(3):1864–1880. doi: 10.1104/pp.16.01803

Figure 9.

Figure 9.

Overexpression of VTC1 or GSH1 rescued the hypersensitivity of myc2-2 mutant to reoxygenation. A, Phenotypes of 4-week-old wild-type (WT), vtc1-1, VTC1-OE, pad2-1, and GSH1-OE plants before submergence (Air) and after 48 h dark submergence (Submergence) followed by a 4 d reoxygenation (Reoxygenation). B, Survival rates (top) and dry weights (bottom) of the WT, vtc1-1, VTC1-OE, pad2-1, and GSH1-OE plants shown in A, following 4 d of reoxygenation. C, Phenotypes of 4-week-old WT, myc2-2, myc2-2 VTC1-OE, VTC1-OE, myc2-2 GSH1-OE, and GSH1-OE plants before submergence (Air) and after 48 h dark submergence (Submergence) followed by a 4 d reoxygenation (Reoxygenation). D, Survival rates (top) and dry weights (bottom) of the WT, myc2-2, myc2-2 VTC1-OE, VTC1-OE, myc2-2 GSH1-OE, and GSH1-OE plants shown in C following 4 d of reoxygenation. The data in B and D are means ± sd (n = 3 independent experiments). For each experiment, 15 plants were used for each genotype. Asterisks indicate significant differences from WT (*P < 0.05; **P < 0.01 by Student’s t test). The differences between myc2-2 and myc2-2 VTC1-OE or myc2-2 GSH1-OE are also significant (**P < 0.01 by Student’s t test). “H” and “L” indicate values that are significantly higher or lower, respectively, in the mutants, double mutants, or transgenic lines than in the WT.