Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2017 Mar 7;11:64. doi: 10.3389/fncel.2017.00064

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2017 Song, Choi and Kim.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

PMC Copyright notice

The expression of pro-inflammatory mediators in Aβ-treated BV2 microglia. (A) The production of nitric oxide (NO) was assessed by Griess reagent assay. Acrp30 reduced the production of NO in Aβ-treated microglia. The increased mRNA level of iNOS (B) and IL-6 (C) and TNF-α (D) were measured by RT PCR. Data are expressed as mean ± SEM. Each experiment was conducted four times per condition. GAPDH served as a control. Differences were considered statistically significant at *p < 0.05. **p < 0.001 (compared to control). Acrp30: Acrp30 5 μg/ml pretreatment, Aβ: Aβ 10 μM treatment for 24 h.