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. 2017 Mar 7;11:64. doi: 10.3389/fncel.2017.00064

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Copyright © 2017 Song, Choi and Kim.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The expression of CD36 and PPAR-γ in Aβ treated BV2 microglia. The mRNA levels of CD36 (A) and PPAR-γ (B) were checked by RT PCR. The reduced mRNA levels of CD36 and PPAR-γ by Acrp30 in Aβ treated microglia were increased by inhibiting AdipoR1. Data are expressed as mean ± SEM. Each experiment was conducted four times per condition. GAPDH served as a control. Differences were considered statistically significant at *p < 0.05 (compared to control). Acrp30: Acrp30 5 μg/ml pretreatment, Aβ: Aβ 10 μM treatment for 24 h, siAdipoR1: siRNA AdipoR1 transfection.