(a, b) Parasternal echocardiography at the onset of heart failure. (a) Short axis view showing pericardiac effusion (red arrow) and all-round ventricular hypertrophy. (b) Motion (M) mode through the left ventricle. The LVDd was 35 mm, left ventricular posterior wall systole (LVDs) 27.6 mm, LVPWD 12.6 mm (196% of normal), and interventricular septal end diastole (IVSd) 13.3 mm (211% of normal). Also, the calculated LVEF was 55%. These findings suggest that diastolic dysfunction due to biventricular hypertrophy resulted in heart failure. (c, d) Parasternal echocardiography 7 days after the initiation of steroid pulse therapy. The LVDd was 40 mm, left ventricular posterior wall systole (LVDs) 23 mm, LVPWD 8.7 mm (133% of normal), and interventricular septal end diastole (IVSd) 10 mm (158% of normal). Also, the calculated LVEF was 80%. Biventricular hypertrophy was improving although still residual, and cardiac contractility was significantly improved.