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. Author manuscript; available in PMC: 2018 Feb 3.
Published in final edited form as: Plant J. 2017 Feb 3;89(3):486–501. doi: 10.1111/tpj.13397

Figure 1.

Figure 1

Enhanced levels of endogenous SA cause the growth defects of cpr5.

(a) Measurement of SA in wild type (Wt), cpr5, sid2 and cpr5 sid2. The levels of free SA and SA glucosides (SAG) in 12-day-old seedlings were analyzed using HPLC. Values represent mean ± SD from three biological replicates.

(b), (c) Seedlings were germinated on 1/2 Linsmaier and Skoog medium for 12 days vertically (b) and horizontally (c). The arrow in (c) indicates the occurrence of necrosis in the cotyledons that is typical of cpr5.

(d) Absolute growth values of seedlings grown as indicated in (b). LOPR, length of primary root; NOLR, number of lateral roots; SFW, shoot fresh weight. Error bars represent SD; n = 24 for each genotype. The P-values were obtained using Student’s t-test for each comparison. **P < 0.001; ***P < 0.0001; ns, not significant.