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. 2017 Mar 3;10:1403–1412. doi: 10.2147/OTT.S111681

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© 2017 Ali et al. This work is published and licensed by Dove Medical Press Limited

The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.

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Pathways of androgen and AR in the pathogenesis of NASH, cirrhosis, and hepatocellular carcinoma.

Note: Visual Art: © 2015 The University of Texas MD Anderson Cancer Center.

Abbreviations: AR, androgen receptor; CCRK, cycle cycle-related kinase; HBV, hepatitis B virus; HCC, hepatocellular carcinoma; HCV, hepatitis C virus; IL, interleukin; MAPK, mitogen-activated protein kinase; NASH, nonalcoholic steatohepatitis; NF-κB, nuclear factor kappa-light-chain-enhancer of activated B cells; STAT 3, signal transducer and activator of transcription 3; TNF, tumor necrosis factor; VEGF, vascular endothelial growth factor; GSK3b, glycogen synthase kinase 3 beta; FFA, free fatty acid; HBVX, hepatitis B virus protein X; TGF1, tumor growth factor 1.