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. 2016 Nov 30;49(11):617–622. doi: 10.5483/BMBRep.2016.49.11.117

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Copyright © 2016 by the The Korean Society for Biochemistry and Molecular Biology

This is an open-access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 4 — Effects of transduced Tat-NQO1 protein in an animal model of ischemia. Gerbils were treated with a single injection of Tat-NQO1 protein (2 mg/kg) before ischemia-reperfusion, and sacrificed after 7 days. Transduced Tat-NQO1 protein was analyzed by immunostaining, using anti-Histidine and DAPI staining. Ischemic neuronal damage was analyzed by NeuN-immunostaining (A). Neuronal cell viability was analyzed by Cresyl violet (CV), F-JB, Iba-1 and GFAP immunostaining. Relative numeric analysis of CV and F-JB, Iba-1, GFAP positive neurons in the CA1 region (B). Scale bar = 18.8 and 50 μm. **P < 0.01, significantly different from the vehicle group.