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. 2017 Feb;29(1):45–56. doi: 10.21147/j.issn.1000-9604.2017.01.06

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Copyright © 2017 Chinese Journal of Cancer Research. All rights reserved.

This work is licensed under a Creative Commons Attribution-Non Commercial-Share Alike 4.0 Unported License. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-sa/4.0/

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Kazrin F promotes migration and invasion of cervical cancer cells. (A) Transwell assays were performed to detect the migration (left panel) and invasion ability (right panel) of HeLa and C33A cells; (B) External cell morphology of HeLa and C33A cells was evaluated with a phase contrast microscope. All photomicrographs were taken at 200× magnification; (C) The protein levels of epithelial-to-mesenchymal transition (EMT)-associated molecules (E-cadherin and vimentin) were determined using a western blot. GAPDH served as a loading control. All data represent the ±s of three independent experiments. *, P<0.05; **, P<0.01; ***, P<0.001.

Inline graphic — Kazrin F promotes migration and invasion of cervical cancer cells. (A) Transwell assays were performed to detect the migration (left panel) and invasion ability (right panel) of HeLa and C33A cells; (B) External cell morphology of HeLa and C33A cells was evaluated with a phase contrast microscope. All photomicrographs were taken at 200× magnification; (C) The protein levels of epithelial-to-mesenchymal transition (EMT)-associated molecules (E-cadherin and vimentin) were determined using a western blot. GAPDH served as a loading control. All data represent the ±s of three independent experiments. *, P<0.05; **, P<0.01; ***, P<0.001.