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. 2017 Jan 5;13(2):581–587. doi: 10.3892/etm.2017.4027

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Copyright: © Gao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Establishment of an experimental mouse model of DN. (A) Relative UACR in each group at different time intervals (weeks 8, 16 and 24). (B) H&E, PAS and Trichrome Masson staining were performed to examine the pathological changes in dissected kidneys from each group. Mice were fed for 24 weeks and experienced significant renal damage as evidenced by the UACR and other parameters presented in Table II. *P<0.05, DN vs. control; ^#P<0.05, DN+L-NAME vs. control. DN, diabetic nephropathy; UACR, urine albumin (µg) to creatinine (µg) ratio; H&E, haemotoxylin and eosin; PAS, Periodic acid-Schiff; L-NAME, NG-nitro-L-Arginine methylester.