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LPA1 mediates VPC31143-induced vascular TXA2 release. A lack of LPA1 but not LPA2 abolishes the VPC31143-elicited elevation in TXA2 production of thoracic aortic segments. LPA1- and LPA2-Ctrl mice showed identical responses and were therefore pooled and referred to as control. **P < 0.01, ****P < 0.0001 vs. before VPC; ####P < 0.0001 vs. control after VPC; 2-way ANOVA with Bonferroni’s post hoc test (n = 6–16).
