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. 2017 Mar 8;5(5):e13169. doi: 10.14814/phy2.13169

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© 2017 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of The Physiological Society and the American Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Acute pancreatitis (see also Table 2). (A) Pancreas to body mass ratio (mg/gm, n = 10 for each group), (B) tissue TBARS (pmole/mg protein, n = 9 for controls, n = 10 for cerulein‐treated groups), and (C) relative mRNA levels (normalized to GAPDH) in control (solid bars) and cerulein‐treated (hatched bars) WT or C1KO mice as marked. For all control groups, n = 9; for all cerulean‐treated groups, n = 10. (D) Left: Western blot of total pancreatic protein probed for CLIC1 (AP1089 antibody, upper panel) or β‐actin (lower panel) from control or cerulein‐treated WT or C1KO mice as marked. Right: Average pancreatic CLIC1 abundance, normalized to β‐actin from control or cerulean‐treated WT mice (con n = 2; cer n = 3). Data presented are means +/‐ SEM. Brackets indicate pairwise comparisons for which P < 0.05.