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. 2009 Sep 10;2(5):325–332. doi: 10.1111/j.1752-8062.2009.00141.x

Figure 5.

Figure 5

HDAC kinase activity is increased three fold in the FHH. Equal amounts of GST‐HDAC5 substrate and GST‐HDACmutant were incubated with nonfailing and failing hearts in the presence of 5 μCi [γ‐32P]‐ATP. (A) Phosphoproteins were resolved on SDS‐PAGE. There was equal loading by corresponding Coomassie staining of the gel (not shown). (B) The signals normalized to the corresponding Coomassie loading are presented in graph form.