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. 2016 Oct 11;8(3):5523–5539. doi: 10.18632/oncotarget.12566

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Copyright: © 2017 Yu et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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a. RHA enhances the transcriptional activity of EWS-FLI1-regulated promoters. The interaction of EWS-FLI1 with RHA affects pre-mRNA processing. b. RHA helicase activity is inhibited by EWS-FLI1. Disrupting RHA binding to EWS-FLI1 by YK-4-279 reduces EWS-FLI1 activity, and restores RHA helicase activity. RHA, RNA helicase A.