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. 2016 Nov 11;14(6):5481–5488. doi: 10.3892/mmr.2016.5936

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Copyright: © Gao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — PD treatment reduces Bax upregulation, Bcl-2 downregulation and cytochrome c release in cortex following middle cerebral artery occlusion. (A) Bax and Bcl-2 protein expression levels in the cortex were detected by western blot. (B) Quantification of Bax protein expression levels relative to GAPDH. (C) Quantification of Bcl-2 protein expression levels relative to GAPDH. (D) Cytosolic cytochrome c levels were determined using a cytochrome c ELISA kit. Data are presented as the mean + standard deviation (n=6 in each group). *P<0.05 vs. sham group; ^#P<0.05 vs. vehicle group. PD, polydatin; Bax, Bcl-associated protein X apoptosis regulator; Bcl-2, B-cell lymphoma 2 apoptosis regulator.