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. 2016 Nov 29;8(2):2372–2380. doi: 10.18632/oncotarget.13676

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Copyright: © 2017 Mehta et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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(A) Cancer recurrence and MPA resistance are not due to loss of PR expression. Seven cervical cancers per group were stained for PR (red) and representative images are shown. Nuclei are shown in blue. A cervical cancer section from K14E7/Pgr^−/− (PRKO) was used as negative control. Dotted lines separate cervical cancer (cc) from surrounding stroma (st). Note that PR is expressed in recurrence (rec.) therapy and prevention group. Scale bar, 50 μm. (B) ERα expression is similar in all cancers. Seven cervical cancers per group were stained for ERα (green) and representative images are shown. Nuclei are shown in blue. A cervical tissue section from Esr1^−/− (ERαKO) was used as negative control. Dotted lines separate cervical cancer (cc) or epithelium (ep) from stroma (st). Scale bar, 50 μm. (C) MCM7 expression is similar in all cancers. Seven cervical cancers per group were stained for MCM7 (red), marker for E7 function and representative images are shown. Nuclei are shown in blue. Normal mouse IgG was used as negative control. Dotted lines separate cervical cancer (cc) from surrounding stroma (st). Scale bar, 30 μm.