A Schematic representation of the B cell receptor (BCR) and TLR pathways. Following BCR engagement, BTK coordinates the activation of IκB kinase (IKK) and leads to NF-κB signaling activation. Similarly, during signaling, MyD88 adaptor proteins stabilize and act as initial platform for IRAK4 recruitment and subsequently IRAK2 recruitment, resulting in NF-κB activation. The downstream activation of STAT3 triggers an autocrine loop, which automantains the MyD88 transcription.