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. 2017 Jan 10;65(4):1278–1292. doi: 10.1002/hep.28947

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© 2016 by the American Association for the Study of Liver Diseases

This article is being made freely available through PubMed Central as part of the COVID-19 public health emergency response. It can be used for unrestricted research re-use and analysis in any form or by any means with acknowledgement of the original source, for the duration of the public health emergency.

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Corresponding peptides (aa 440‐448) on TUCH and Ro1845 have no effect on rotavirus binding and replication. (A) Amino acids 440‐448 of RRV, TUCH, and Ro1845. (B, C) Treatment of mouse cholangiocytes with 1 mM MRTRVSGLY and VRTRISGLY had no effect on binding or replication in RRV, TUCH, or Ro1845. In all strains, DGEA (positive control) significantly inhibited both viral binding and replication compared with media‐only control. Treatment with TRTRVSRLY significantly reduced RRV binding and viral yield with no effect on other rotavirus strains. *P < 0.05 versus serum‐free media pretreatment.