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. 2017 Mar 23;18:251. doi: 10.1186/s12864-017-3634-5

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Conceptual figure showing how the multiallelic overdominance model (left) and multilocus pseudooverdominance model (right) work to make the parents (a) produce the marker and sex segregation patterns observed in the S5 (b) and S3 populations (c). Potentially lethal alleles are coded as small letters (e.g., Z-a-b-c-d) in contrast to alleles of with no adverse effect (Z-A-B-C-D). Sex-marker genotype combinations not observed and potentially being caused by incompatible/lethal genotypes are crossed over. Note: The particular examples in this figure show a sex-neutral version of the model where the W- as well as Z-gametologs may be associated with lethal alleles