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. 2016 Dec 16;15(2):712–718. doi: 10.3892/mmr.2016.6055

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Copyright: © Zhao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Hyperlipidemia-induced hippocampal neuronal apoptosis of apoE(−/−) mice may be controlled via the Bcl-2/Bax-caspase-3 pathway. Immunohistochemistry of Bcl-2, Bax, and caspase-3 in hippocampus CA1 and CA3 of apoE(−/−) mice fed with ND or HFD for 12 weeks (magnification, ×400). Brown particles in the cytoplasm indicate protein expression. ND, normal diet; HFD, high-fat diet; Bcl-2, B-cell lymphoma 2; Bax, Bcl-2-associated X protein.