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. 2016 Dec 29;15(2):613–626. doi: 10.3892/mmr.2016.6083

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Copyright: © Xiang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Comparison of liver histology in mice with DMN-induced liver cirrhosis. Mice were injected with DMN or normal saline for 4 weeks. Following 4 weeks of DMN treatment, mice were subsequently treated with YGD or HGF for a further 4 weeks. Liver tissues were stained with (A) hematoxylin and eosin and (B) Masson's trichrome. Liver damage induced by DMN was alleviated by YGD or HGF treatment. Magnification, ×100; scale bar=200 µm. YGD, Yi Guan Jian decoction; HGF, hepatocyte growth factor; DMN, dimethylnitrosamine; C, control; M, model; NC, negative control; W, week.

Figure 1. — Comparison of liver histology in mice with DMN-induced liver cirrhosis. Mice were injected with DMN or normal saline for 4 weeks. Following 4 weeks of DMN treatment, mice were subsequently treated with YGD or HGF for a further 4 weeks. Liver tissues were stained with (A) hematoxylin and eosin and (B) Masson's trichrome. Liver damage induced by DMN was alleviated by YGD or HGF treatment. Magnification, ×100; scale bar=200 µm. YGD, Yi Guan Jian decoction; HGF, hepatocyte growth factor; DMN, dimethylnitrosamine; C, control; M, model; NC, negative control; W, week.