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. 2016 Dec 19;124(4):1330–1338. doi: 10.1213/ANE.0000000000001755

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Copyright © 2016 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the International Anesthesia Research Society.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Figure 6. — Expression level of M1 and M2 receptors in the prefrontal cortex (PFC), caudate putamen (CPU), and ventral tegmental area (VTA) of mice with peripheral nerve injuries. A, Expression levels of M2 in the PFC were decreased at D14 after nerve injury. (**P < .01, n = 4 for sham, n = 5 for injury). B, Expression levels of M2 in the CPU were decreased at D14 after nerve injury (**P < .01). C, Expression levels of M2 in the VTA were decreased at D14 after nerve injury (**P < .01). D, Expression levels of M1 in the PFC were decreased at D14 after nerve injury (**P < .01). E, Expression levels of M1 in the CPU were decreased at D14 after nerve injury (*P < .05). F, Expression levels of M1 in the VTA were decreased at D14 after nerve injury (**P < .01).