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. 2017 Mar 29;5(6):e13202. doi: 10.14814/phy2.13202

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© 2017 Universidad De Buenos Aires. Physiological Reports published by Wiley Periodicals, Inc. on behalf of The Physiological Society and the American Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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NO metabolites (Nitrates and Nitrites, NOx) production in renal cortex. NS: normal sodium intake; HS: high sodium intake; control: rats not treated with the D₁‐like receptor antagonist SCH 23390; SCH 23390: rats treated with the D₁‐like receptor antagonist. Two way ANOVA showed a statistically significant interaction (P < 0.01) between the effects of high salt intake and SCH 23390 treatment on NOx production in renal cortex. *P < 0.01 HS versus NS; & P < 0.001 HS + SCH 23390 versus HS. Results are expressed as mean ± SEM (n = 5).