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. Author manuscript; available in PMC: 2018 Feb 7.
Published in final edited form as: Cell Metab. 2017 Feb 7;25(2):386–399. doi: 10.1016/j.cmet.2017.01.002

Figure 4. Actβ signals through Babo to affect AKH signaling in the fat body.

Figure 4

(A) Glycemic levels (glucose + trehalose) in 3rd instar larvae with systemic Actβ knockdown (left, Actin-Gal4/UAS-Actβ-RNAi) or knockout (right, actβ ed80 or Actβ −/−) fed a standard or high-sugar diet. (B, C) Glycemic levels in CG > Actβ (CG-Gal4/+; UAS-Actβ/+) larvae (B) or CG > Actβ larvae with gene knockdown (CG-Gal4, tub-Gal80ts/+; UAS-Actβ/UAS-RNAi) (C) (n=4, 40 larvae). (D) Immunoblots in freshly isolated fat bodies of Actβ−/− larvae fed a high-sugar diet (left) or CG > Actβ larvae fed normal food (right). Data are presented as means ± SEM. * p < 0.05.