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. 2016 Nov 28;6(3):311–316. doi: 10.1242/bio.021410

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© 2017. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 1. — Parecoxib inhibits cell proliferation of GBM cells. (A) The relative cell viability of U251 cells was determined by MTT assay. U251 cells were treated with 0, 20, 50, 100, and 200 μM of parecoxib for 24 and 48 h. Mean±s.d., n=3, *P<0.05, **P<0.01 compared with 0 μM of parecoxib at 24 h; #P<0.05, ##P<0.01 compared with 0 μM of parecoxib at 48 h. (B) The relative cell viability of U343 cells was determined by MTT assay. U343 cells were treated with 0, 20, 50, 100, and 200 μM of parecoxib for 24 and 48 h. Mean±s.d., n=3, *P<0.05 compared with 0 μM of parecoxib at 24 h; #P<0.05, ##P<0.01 compared with 0 μM of parecoxib at 48 h. (C) The BrdU incorporation of U251 cells was determined by BrdU assay. U251 cells were treated with 0, 100, and 200 μM of parecoxib for 24 and 48 h. Mean±s.d., n=3, *P<0.05 compared with 0 μM of parecoxib at 24 h; #P<0.05 compared with 0 μM of parecoxib at 48 h. (D) The BrdU incorporation of U343 cells was determined by BrdU assay. U343 cells were treated with 0, 100, and 200 μM of parecoxib for 24 and 48 h. Mean±s.d., n=3, *P<0.05 compared with 0 μM of parecoxib at 24 h; #P<0.05 compared with 0 μM of parecoxib at 48 h.